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Clinton Jones, Ph.D.

Transcriptional regulation, neuro-degeneration, effect of stress

Degree: Ph.D.
Broad Expertise/Interests: Neurotropic herpesviruses and neurobiology
Equipment/Techniques: Animal models, genomics, gene expression, cell death,  immunohistochemistry
Seeking Collaborators with Skill Sets: RNA-Seq, source of neurons
Neuroscience Research Interests: Neurotropic herpesviruses enter CNS and induce encephalitis.  Identification of viral genes that are expressed in neurons that are latently infected.  Understanding stress-induced reactivation from latency. Identification of cellular genes that gb-[[[[[[[ett latency in neurons.    
Funding in the Last Three Years:
  • 2 USDA grant; $500,000 each
  • NIH grant: $420,000
Publication Highlights:

Workman, A. L. Zhu, B.N. Keel, T.P.L. Smith, and C. Jones.  The Wnt signaling pathway is differentially expressed during the bovine herpesvirus 1 latency-reactivation cycle: evidence that two protein kinases associated with neuronal survival (Akt3 and bone morphogenetic protein receptor 2) are expressed at higher levels during latency.  2018. J of Virology 92: e01937-17.

Jones, C. and E.M. Scholar.  2016. Viral Induced Encephalitis, Neuroimmune Pharmacology, T. Iketsu, H.E. Gendelman edts, Springer, 2nd Edition, 437-449.

 Zhu, L., A. Workman, and C. Jones.  2017. A potential role for a b-catenin coactivator (high mobility group AT-hook 1 protein) during the latency-reactivation cycle of bovine herpesvirus 1.  J of Virology: 91: e02132-16.